The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease” - Archive ouverte HAL Access content directly
Journal Articles Free Radical Biology and Medicine Year : 2010

The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”

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cea-02168439 , version 1 (28-06-2019)

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Carole Escartin, Emmanuel Brouillet. The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”. Free Radical Biology and Medicine, 2010, 49 (2), pp.144-146. ⟨10.1016/j.freeradbiomed.2010.04.009⟩. ⟨cea-02168439⟩
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