The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”

Carole Escartin 1 Emmanuel Brouillet 1, *
* Corresponding author
1 LMN - Laboratoire des Maladies Neurodégénératives - UMR 9199
MIRCEN - Service MIRCEN : DRF/JACOB, UP11 - Université Paris-Sud - Paris 11, CNRS - Centre National de la Recherche Scientifique : UMR 9199
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Submitted on : Friday, June 28, 2019 - 4:45:12 PM
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Carole Escartin, Emmanuel Brouillet. The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”. Free Radical Biology and Medicine, Elsevier, 2010, 49 (2), pp.144-146. ⟨10.1016/j.freeradbiomed.2010.04.009⟩. ⟨cea-02168439⟩

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