The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide
improve the behavioral phenotype and brain pathology in a transgenic mouse model
of Huntington disease”

Carole Escartin; Emmanuel Brouillet

doi:10.1016/j.freeradbiomed.2010.04.009

Journal articles

The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”

Carole Escartin ¹ Emmanuel Brouillet ^{1, *}

* Corresponding author

1 LMN - Laboratoire des Maladies Neurodégénératives - UMR 9199

MIRCEN - Service MIRCEN : DRF/JACOB, CNRS - Centre National de la Recherche Scientifique : UMR 9199

Document type :

Journal articles

Domain :

Life Sciences [q-bio] / Neurons and Cognition [q-bio.NC] / Neurobiology

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https://hal-cea.archives-ouvertes.fr/cea-02168439
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Submitted on : Friday, June 28, 2019 - 4:45:12 PM
Last modification on : Monday, December 13, 2021 - 9:16:09 AM

The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”

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The Nrf2 pathway as a potential therapeutic target for Huntington disease. A commentary on “Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington disease”

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